Publication
Genetic alterations of ALK in high-risk neuroblastoma patients: a SIOPEN study
| dc.contributor.author | Bellini, Angela | |
| dc.contributor.author | Bernard, Virginie | |
| dc.contributor.author | Ambros, Inge | |
| dc.contributor.author | Ambros, Peter F. | |
| dc.contributor.author | de Preter, Katleen | |
| dc.contributor.author | Combaret, Valérie | |
| dc.contributor.author | Beiske, Klaus | |
| dc.contributor.author | Jeison, Marta | |
| dc.contributor.author | Marques, Barbara | |
| dc.contributor.author | Morini, Martina | |
| dc.contributor.author | Mazzocco, Katia | |
| dc.contributor.author | Defferrari, Raffaella | |
| dc.contributor.author | Betts, David | |
| dc.contributor.author | Martinsson, Tommy | |
| dc.contributor.author | Mühlethaler-Mottet, Annick | |
| dc.contributor.author | Noguera, Rosa | |
| dc.contributor.author | Font de Mora, Jaime | |
| dc.contributor.author | Vicha, Ales | |
| dc.contributor.author | Ladenstein, Ruth | |
| dc.contributor.author | Valteau-Couanet, Dominique | |
| dc.contributor.author | Rossing, Caroline Maria | |
| dc.contributor.author | Bown, Nick | |
| dc.contributor.author | Tweddle, Deborah | |
| dc.contributor.author | Avigad, Smadar | |
| dc.contributor.author | Lapouble, Eve | |
| dc.contributor.author | Chicard, Mathieu | |
| dc.contributor.author | Leprovost, Nada | |
| dc.contributor.author | Clement, Nathalie | |
| dc.contributor.author | Baulande, Sylvain | |
| dc.contributor.author | Pierron, Gaelle | |
| dc.contributor.author | Irene, Jimenez | |
| dc.contributor.author | Jaydutt, Bhalshankar | |
| dc.contributor.author | Delattre, Olivier | |
| dc.contributor.author | Michon, Jean | |
| dc.contributor.author | Schleiermacher, Gudrun | |
| dc.date.accessioned | 2019-02-19T16:30:46Z | |
| dc.date.available | 2019-02-19T16:30:46Z | |
| dc.date.issued | 2018-10-10 | |
| dc.description.abstract | Background: In neuroblastoma (NB), the ALK receptor tyrosine kinase can be constitutively activated either through genomic amplification or activating point mutations. We studied ALK genetic alterations in high-risk NB patients to determine their frequency and prognostic impact. | pt_PT |
| dc.description.version | N/A | pt_PT |
| dc.identifier.uri | http://hdl.handle.net/10400.18/5900 | |
| dc.language.iso | eng | pt_PT |
| dc.peerreviewed | yes | pt_PT |
| dc.rights.uri | http://creativecommons.org/licenses/by/4.0/ | pt_PT |
| dc.subject | Neuroblastoma | pt_PT |
| dc.subject | ALK | pt_PT |
| dc.subject | Tumor Genomics | pt_PT |
| dc.subject | Doenças Genéticas | pt_PT |
| dc.title | Genetic alterations of ALK in high-risk neuroblastoma patients: a SIOPEN study | pt_PT |
| dc.type | conference object | |
| dspace.entity.type | Publication | |
| oaire.citation.conferencePlace | Jerusalém, Israel | pt_PT |
| oaire.citation.title | SIOPEN – International Collaboration for Neuroblastoma Research, Annual General Meeting 2018 - 30-year anniversary Conference, 10 Oct 2018 | pt_PT |
| rcaap.rights | openAccess | pt_PT |
| rcaap.type | conferenceObject | pt_PT |
Files
Original bundle
1 - 1 of 1
Loading...
- Name:
- Abstract ALK SIOPEN SIOPEM Oct2018_submittedVers.pdf
- Size:
- 266.82 KB
- Format:
- Adobe Portable Document Format
License bundle
1 - 1 of 1
No Thumbnail Available
- Name:
- license.txt
- Size:
- 1.71 KB
- Format:
- Item-specific license agreed upon to submission
- Description:
