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Assessment of indoor air exposure among newborns and their mothers: Levels and sources of PM10, PM2.5 and ultrafine particles at 65 home environments

2020-09Journal article Restricted access
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dc.contributor.authorMadureira, Joana
dc.contributor.authorSlezakova, Klara
dc.contributor.authorCosta, Carla
dc.contributor.authorPereira, Maria Carmo
dc.contributor.authorTeixeira, João Paulo
dc.date.accessioned2021-03-04T12:04:39Z
dc.date.available2021-03-04T12:04:39Z
dc.date.issued2020-09
dc.description.abstractSignificant efforts have been directed towards addressing the adverse health effects of atmospheric particles, emphasizing the relevance of indoor exposure. Homes represent an indoor environment where human spend the majority of their time. Thus, the objective of this work was to concurrently assess different matrix of indoor particles considering both mass (PM10, PM2.5) and number (N20-1000) concentrations in indoor and outdoor air of homes (n = 65). Real-time measurements (PM10, PM2.5, UFP) were conducted simultaneously during 48 h in dwellings situated in Oporto, Portugal. In 75% of homes, indoor PM2.5 (mean = 53 μg m-3) exceeded limit of 25 μg m-3, for PM10 (mean = 57 μg m-3) 41% of homes demonstrated average levels higher than 50 μg m-3, thus indicating potential risks. Indoor PM10 was mostly (82-99%) composed of PM2.5, both PM were highly correlated (|rs|>0.9655), thus suggesting the similar origin. Indoor PM originated from infiltrations of outdoor emissions; ∼70% of homes exhibited indoor to outdoor (I/O) ratio < 1. On the contrary, UFP indoors (mean = 13.3 × 103 # cm-3) were higher than outdoors (mean = 10.0 × 103 # cm-3). Indoor UFP spatially varied as follows: kitchens > living rooms > bedrooms. UFP indoors were poorly correlated (|rs| = 0.456) with outdoor concentrations, I/O ratios showed that indoor UFP predominantly originated from indoor emission sources (combustions). Therefore, in order to reduce exposure to UFP and protect public health, the primary concerns should be focused on controlling emissions from indoor sources.pt_PT
dc.description.abstractHighlights: Indoor UFP were poorly correlated with PM (|rs|<0.552–0.594); Indoor UFP were approximately 30% higher indoors that outdoors (|rs| = 0.456); Indoor UFP mainly originated from indoor sources (mean I/O = 1.59, range 0.27–6.67); Indoor PM2.5 mostly resulted from outdoor emissions infiltrations (mean I/O = 0.88); PM2.5 and PM10 exceeded WHO daily limits in 75% and 41% of homes, respectively.pt_PT
dc.description.sponsorshipThis work was supported by FCT and FAPESP (FAPESP/19914/2014) and by Base Funding e UIDB/00511/2020 of the Laboratory for Process Engineering, Environment, Biotechnology and Energy e LEPABE e funded by national funds through the FCT / MCTES (PIDDAC). Joana Madureira and Carla Costa are supported by FCT (SFRH/BPD/115112/2016 and SFRH/BPD/96196/2013 grants, respectively).pt_PT
dc.description.versioninfo:eu-repo/semantics/publishedVersionpt_PT
dc.identifier.citationEnviron Pollut. 2020 Sep;264:114746. doi: 10.1016/j.envpol.2020.114746. Epub 2020 May 7pt_PT
dc.identifier.doi10.1016/j.envpol.2020.114746pt_PT
dc.identifier.issn0269-7491
dc.identifier.urihttp://hdl.handle.net/10400.18/7309
dc.language.isoengpt_PT
dc.peerreviewedyespt_PT
dc.publisherElsevierpt_PT
dc.relationLaboratory for Process Engineering, Environment, Biotechnology and Energy
dc.relationEarly life exposure to atmospheric air pollutants and potential DNA damage: Consequences on development and childhood outcomes APPEAL
dc.relationImpact of transplacental exposure to tobacco smoke in the DNA of newborn. Evaluation of genetic damage and epigenetic changes. Evolution throughout childhood.
dc.relation.publisherversionhttps://www.sciencedirect.com/science/article/abs/pii/S0269749120310551?via%3Dihubpt_PT
dc.rights.urihttp://creativecommons.org/licenses/by-nc-nd/4.0/pt_PT
dc.subjectAir Pollutantspt_PT
dc.subjectAir Pollution, Indoorpt_PT
dc.subjectEnvironmental Monitoringpt_PT
dc.subjectFemalept_PT
dc.subjectHumanspt_PT
dc.subjectInfant, Newbornpt_PT
dc.subjectMotherspt_PT
dc.subjectParticle Sizept_PT
dc.subjectParticulate Matterpt_PT
dc.subjectPortugalpt_PT
dc.subjectAr e Saúde Ocupacionalpt_PT
dc.titleAssessment of indoor air exposure among newborns and their mothers: Levels and sources of PM10, PM2.5 and ultrafine particles at 65 home environmentspt_PT
dc.typejournal article
dspace.entity.typePublication
oaire.awardTitleLaboratory for Process Engineering, Environment, Biotechnology and Energy
oaire.awardTitleEarly life exposure to atmospheric air pollutants and potential DNA damage: Consequences on development and childhood outcomes APPEAL
oaire.awardTitleImpact of transplacental exposure to tobacco smoke in the DNA of newborn. Evaluation of genetic damage and epigenetic changes. Evolution throughout childhood.
oaire.awardURIinfo:eu-repo/grantAgreement/FCT/6817 - DCRRNI ID/UIDB%2F00511%2F2020/PT
oaire.awardURIinfo:eu-repo/grantAgreement/FCT//SFRH%2FBPD%2F115112%2F2016/PT
oaire.awardURIinfo:eu-repo/grantAgreement/FCT//SFRH%2FBPD%2F96196%2F2013/PT
oaire.citation.startPage114746pt_PT
oaire.citation.titleEnvironmental Pollutionpt_PT
oaire.citation.volume264pt_PT
oaire.fundingStream6817 - DCRRNI ID
project.funder.identifierhttp://doi.org/10.13039/501100001871
project.funder.identifierhttp://doi.org/10.13039/501100001871
project.funder.identifierhttp://doi.org/10.13039/501100001871
project.funder.nameFundação para a Ciência e a Tecnologia
project.funder.nameFundação para a Ciência e a Tecnologia
project.funder.nameFundação para a Ciência e a Tecnologia
rcaap.embargofctAcesso de acordo com política editorial da revista.pt_PT
rcaap.rightsembargoedAccesspt_PT
rcaap.typearticlept_PT
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relation.isProjectOfPublication.latestForDiscovery2f8cdc25-2875-4f26-8876-a08085c7f459

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