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Towards a better assessment of the mercury-selenium antagonism in fish

dc.contributor.authorMendes Ribeiro, Mariana
dc.contributor.authorZephyr, N.
dc.contributor.authorSilva, José Armando
dc.contributor.authorDamion, M.
dc.contributor.authorGuérin, T.
dc.contributor.authorCastanheira, Isabel
dc.contributor.authorLeufroy, A.
dc.contributor.authorJitaru, P.
dc.date.accessioned2022-07-07T16:39:49Z
dc.date.available2022-07-07T16:39:49Z
dc.date.issued2021-07-02
dc.description.abstractThe fish were killed by cutting the spinal cord in t 0 t 1 t 2 and t 3 All the data related to the fish growth were recorded similar growth was observed for all exposure conditions. The levels of Hg T and Se T in the fish diet were also measured by ICP MS. Hg T in control and MeHg spiked diets were 0.05 ±0.002 and 0.24 ±0.01 mg/kg, respectively, whereas Se T were 0.95 ±0.02 mg/kg and 5.41 ±0.36 mg/kg in the control and the spiked diets. Se saturation ( p = 0.30) occurs after two months of fish be exposed throught diet to SeMet solely. Quasilinear (R 2  0.99) bioaccumulation of Hg in the fish exposed individualy to MeHg during 3 months. Threshold (p = 0.30) in total Hg level is reached in fish exposed simultaneously to MeHg and SeMet. ↓ of Hg burden  18%) after two months of fish exposure to SeMet and no MeHg. Possible Hg detoxification due to Se supplementation.pt_PT
dc.description.sponsorshipFCT, MERSEL-FISH - 2018DAN1568pt_PT
dc.description.versionN/Apt_PT
dc.identifier.urihttp://hdl.handle.net/10400.18/8087
dc.language.isoengpt_PT
dc.peerreviewedyespt_PT
dc.subjectFishpt_PT
dc.subjectMercurypt_PT
dc.subjectSeleniumpt_PT
dc.subjectComposição dos Alimentospt_PT
dc.subjectSegurança Alimentarpt_PT
dc.titleTowards a better assessment of the mercury-selenium antagonism in fishpt_PT
dc.typeconference object
dspace.entity.typePublication
oaire.citation.conferencePlace(Online)pt_PT
oaire.citation.titleVirtual Food Science Symposium, AOAC Europe/SFC, 1-2 July 2021pt_PT
rcaap.rightsclosedAccesspt_PT
rcaap.typeconferenceObjectpt_PT

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